Neidhart and Matthias conclude that the origin of this condition might be sought for in the disturbance of the nerve-centers.

After reviewing all these theories, Salzer states that in his case the patient was previously healthy and never had suffered the slightest hemorrhage in any part, and he therefore rejects the theory of extravasation.

He is inclined to suppose that the priapism was due to the stimulation of the nervi erigentes, brought about either by anatomic change in the nerves themselves, or by pressure upon them by enlarged lumbar glands, an associate condition of leukemia.
Burchard reports a most interesting case of prolonged priapism in an English gentleman of fifty-three.

When he was called to see the man on July 15th he found him suffering with intense pain in the penis, and in a state of extreme exhaustion after an erection which had lasted five hours uninterruptedly, during the whole of which time the organ was in a state of violent and continuous spasm.

The paroxysm was controlled by 3/4 grain morphin and 1/50 grain atropin.